亚洲免费av电影一区二区三区,日韩爱爱视频,51精品视频一区二区三区,91视频爱爱,日韩欧美在线播放视频,中文字幕少妇AV,亚洲电影中文字幕,久久久久亚洲av成人网址,久久综合视频网站,国产在线不卡免费播放

        ?

        Global Stability of An Eco-epidemiological Model with Beddington-DeAngelis Functional Response and Delay

        2020-01-07 06:27:54BAIHongfangXURui

        BAI Hong-fang, XU Rui

        (1.Faculty of Science and Technology, University of Macau, Macau Special Administrative Region,China; 2.Complex Systems Research Center, Shanxi University, Taiyuan, 030006, China)

        Abstract: In this paper, an eco-epidemiological model with Beddington-DeAngelis functional response and a time delay representing the gestation period of the predator is studied.By means of Lyapunov functionals and Laselle’s invariance principle,sufficient conditions are obtained for the global stability of the interior equilibrium and the disease-free equilibrium of the system, respectively.

        Key words:Eco-epidemiological model;delay;Laselle’s invariance principle;global stability

        §1. Introduction

        It is necessary to study the effect of epidemiological parameters in the ecological domain from mathematical as well as ecological point of view. As a new branch in mathematical biology, eco-epidemiology merges ecology and epidemiology to understand the dynamics of disease propagation on the prey-predator population.

        The disease factor in predator-prey system was first introduced by Anderson and May [1].Since this pioneering work, great attention has been paid to the modeling and analysis of eco-epidemiological systems recently, and an increasing number of works have been devoted to the study of the relationships between demographic processes among different populations and diseases [2-12]. Such as, Zhang et al. [8]studied the following delayed eco-epidemiological model with Holling type-I response function

        where x(t),S(t),I(t) denote the densities of the prey, the susceptible predator and the infected predator population, respectively.

        The functional response is a key element in all predator-prey interactions. The functional response refers to the number of prey eaten per predator per unit time as a function of prey density.System (1.1) assumes that the per capita rate of predation depends on the prey numbers only.But there is growing explicit biological and physiological evidence that in some situations,especially when predators have to research for food(and therefore have to share or compete for food),a more realistic model should be based on a functional response which is predator-dependent.Such as , ”ratio-dependent” theory [13-15], and in some cases, the Beddington-DeAngelis type functional response performed even better. The Beddington-DeAngelis functional responsewas introduced by Beddington-DeAngelis et al. [16-17]. It is similar to the well-known Holling type II functional response but has an extra term nS(t)in the denominator which models mutual interference between the susceptible predator. When m > 0, n = 0, the Beddington-DeAngelis functional response is simplified to Holing type II functional response.And when m=0, n>0, it expresses a saturation response.

        Motivated by the works of Zhang et al. [8]and Beddington-DeAngelis et al. [16-17], in this paper, we are concerned with the combined effects of the disease transmission, Beddington-DeAngelis functional response and time delay due to the gestation of predator on the global dynamics of a predator-prey system. To this end, we consider the following eco-epidemiological model with delay:

        where x(t), S(t) and I(t) denote the densities of the prey, the susceptible predator and the infected predator population, respectively. r is the intrinsic growth rate of prey population without disease, r/a11is the environmental carrying capacity, a12is the capturing rate of the susceptible predators, a21/a12is the conversion rate of nutrients into the reproduction of the susceptible predators by consuming prey, β is the disease transmission coefficient, r1is the natural death rate of the susceptible predators, r2is the natural and disease-related mortality rate of the infected predator. Here, r1≤r2. τ is a time delay representing a duration of τ time units elapses when an individual prey is killed and the moment when the corresponding addition is made to the predator population. All the parameters are positive.

        We denote by C the Banach space of continuous functions φ:[?τ,0]→R3with norm

        where φ=(φ1, φ2, φ3)∈C. Further, let

        The initial conditions for system (1.3) take the form

        where φ=(φ1, φ2, φ3)∈C+.

        The organization of this paper is as follows. In sec. 2,we present some preliminaries,such as the positivity, and the equilibria of system (1.2). In sec. 3, we consider about the permanence of system (1.2) by using the persistence theory on infinite dimensional systems developed by Hale and Waltman [18]. In sec. 4, by means of suitable Lyapunov functionals and Lasalle’s invariance principle, we establish sufficient conditions for the global asymptotic stability of the interior equilibrium and the disease-free equilibrium of system (1.2). Finally, the paper ends with a summary and discussion.

        §2. Preliminaries

        In this section, we show the positivity of solutions and the equilibria of system (1.2).

        2.1 Positivity of solutions

        Theorem 2.1Suppose that (x(t),S(t),I(t)) is a solution of system (1.2) with initial conditions (1.3). Then x(t)>0, S(t)>0 and I(t)>0 for all t ≥0.

        ProofFrom the first and last equation of system (1.2), we have

        Hence, x(t) and I(t) are positive.

        To show that S(t)is positive on[0,∞),suppose that there exists t2>0 such that S(t2)=0,and S(t)>0 for t ∈[0,t2). Then ˙S(t2)≤0. From the second equation of (1.2), we have

        which is a contradiction.

        Next, we will give the equilibria of system (1.2).

        2.2 equilibriaEquilibria of system (1.2) are obtained by setting the right side of three equations of (1.2) to zeros. Doing this, we get four equilibria in general.

        (i) The trivial equilibrium E0=(0,0,0).

        (ii) The predator-extinction equilibrium E1=(r/a11,0,0).

        (iii) The disease-free equilibrium E2=(x2,S2,0), where

        Obviously, if

        (H1) a21?mr1>a11r1/r,

        then x2>0, S2>0.

        (iv) The interior equilibrium E?=(x?,S?,I?), where

        It can be seen that if

        then system (1.2) has a interior equilibrium E?.

        §3. Permanence

        In this section, we consider about the permanence of system (1.2).

        Definition 3.1System (1.2) is said to be permanent (uniformly persistent) if there are positive miand Mi(i=1,2,3)such that each positive solution(x(t),S(t),I(t))of system(1.2)satisfies

        Let X be a complete metric space with metric d. Suppose that T is a continuous semiflow on X, that is, a continuous mapping T :[0,+∞)×X →X with the following properties

        where Ttdenotes the mapping from X to X given by Tt(x)=T(t,x).

        The distance d(x,Y) of a point x ∈X from a subset Y of X is defined by d(x,Y)=infy∈Yd(x,y).

        Recall that the positive orbit γ+(x) through x is defined asand its ω?limit set is ω(x)=Define Ws(A)the strong stable set of a compact invariant set A as

        Suppose that X0is open and dense in X and X0∪X0= X, X0?. Moreover, the C0-semigroup T(t) on X satisfies

        Let Tb(t)=T(t)|X0and Abbe the global attractor for Tb(t).

        Lemma 3.1rm(Hale and Waltman[19])Suppose that T(t)satisfies(3.1). If the following hold

        (i) there is a t0≥0 such that T(t) is compact for t>t0;

        (ii) T(t) is point dissipative in X; and

        Then X0is a uniform repeller with respect to X0, that is, there is an ε > 0 such that for any x ∈X0,

        In order to study the permanence of system (1.2), we also need following result.

        Lemma 3.2There are positive constants M1and M2such that for any positive solution(x(t), S(t), I(t)) of system (1.2) with initial conditions (1.3),

        ProofLet(x(t),S(t),I(t))be any positive solution of system(1.2)with initial conditions(1.3). Set

        Calculating the derivative of V(t) along positive solution of system (1.2), we get

        where M1=which yields≤M1. If we choose M2= a21M1/a12, then(3.2) follows. This complete the proof.

        We are now in a position to state and prove our result on the permanence of system (1.2).

        Theorem 3.1If βS2>r2and (H1) hold, then system (1.2) is permanent.

        ProofLet C+([?τ,0],R3+)denote the space of continuous functions mapping[?τ,0]into R3+. Define

        Denote C0=C1∪C2, X =C+([?τ,0],R3+) and C0=intC+([?τ,0],R3+).

        In the following, we verify the conditions in Lemma 3.1 are satisfied. By the definition of C0and C0, it is easy to see that C0and C0are positively invariant and the conditions (i) and(ii) in Lemma 3.1 are clearly satisfied. Thus, we need only to show that the conditions (iii)and (iv) hold. Clearly, system (1.2) possesses two constant solutions in C0:corresponding, respectively, to x(t) = r/a11, S(t) = 0, I(t) = 0 and x(t) = x2, S(t) = S2,I(t)=0.

        We now verify the condition (iii) of Lemma 3.1. If (x(t),S(t),I(t)) is a solution of system(1.2) initiating from C1, then ˙x(t)=rx(t)?a11x2(t), which yields x(t)→r/a11as t →+∞. If(x(t),S(t),I(t)) is a solution of system (1.2) initiating from C2with φ1(θ) > 0 and φ2(θ) > 0,then we have

        Using Lemma 3.1 and Lemma 3.2, it is not difficult to prove that if (H1) holds, then system(3.3) is uniformly persistent. Noting that C1∩C2=?, it follows that the invariant setsandare isolated. Hence,is isolated and is an acyclic covering satisfying the condition(iii) in Lemma 3.1.

        §4. Global Stability

        In this section, we give some sufficient conditions for global stability of the interior equilibrium E?and the disease-free equilibrium E2, respectively. The method of proofs is to use global Lyapunov functional and Lasalle’s invariance principle.

        Theorem 4.1If the interior equilibrium E?of system (1.2) exists, then E?is globally asymptotically stable provided that

        ProofAssume that (x(t),S(t),I(t)) is any positive solution of system (1.2) with initial conditions (1.3). Denote φ(x(t),S(t))=Define

        Calculating the derivative of V11(t) along positive solutions of system (1.2), it follows that

        On substituting rx??a11x?2?a12φ(x?,S?) = 0, a21φ(x?,S?)?r1S??βS?I?= 0 and βS?=r2into Eq. (4.2), we derive that

        Define

        Then

        Set V1(t)=V11(t)+V12(t). It follows from (4.1) (4.4) and (4.5) that

        Collecting terms of Eq. (4.6), we get

        On substituting a21φ(x?,S?)=r1S?+βS?I?and βS?I?=r2I?into Eq. (4.7), we derive that

        Noting that

        we derive from (4.8) that

        Because (H2) holds, there is a constant T >0 such that if t ≥T, x(t)>r/(2a11). In this case,we have that, for t ≥T,

        with equality if and only if x = x?. Note that the function f(x) = x ?1 ?ln x is always non-negative for any x>0, and f(x)=0 if and only if x=1. Therefor, we have that if t ≥T,˙V1(t) ≤0, which equality if and only if x = x?,S = S?. We now look for the invariant subset M within the set

        Since x = x?,S = S?on M, we obtain from the second equation of system (1.2) that 0 =which yields I =I?. Hence, the only invariant set in M = {(x,S,I) := 0} is M = (x?,S?,I?). Therefore, the global asymptotic stability of E?follows from Lasalle’s invariance principle for delay differential systems[18]. This completes the proof.

        Theorem 4.2If βS2?r2<0 and (H2) hold, the disease-free equilibrium E2(x2,S2,0) is globally asymptotically stable.

        ProofAssume that (x(t),S(t),I(t)) is any positive solution of system (1.2) with initial conditions (1.3). Denote

        Calculating the derivative of V21(t) along positive solutions of system (1.2), it follows that

        On substituting rx2?a11?a12φ(x2,S2)=0 and a21φ(x2,S2)=r1S2into Eq. (4.13), we derive that

        Define

        Then

        Collecting terms of Eq. (4.17), we get

        On substituting a21φ(x2,S2)=r1S2into Eq. (4.18), we derive that

        Noting that

        we derive from (4.19) that

        Hence, if follow from (4.21) that if βS2?r2< 0 and (H2) hold, then≤0 for t ≥T,with equality if and only if x = x2,S = S2,I = 0. It shows that the only invariant set in M = {(x,S,I) := 0} is M = {(x2,S2,0)}. Using Lasalle’s invariance principle for delay differential systems [18]. This completes the proof.

        §5. Discussion

        In this paper, we have investigated the global dynamics of a delayed predator-prey model with a transmissible disease spreading among the predator population. We noted that system(1.2) has no intra-specific competition terms in the second and the third equations. In this situation, under what conditions will the global stability of a feasible equilibrium of system(1.3) persists independent of the time delay due to the gestation of the predator? To solve this problem, by using Lyapunov functionals and Laselle’s invariance principle, we established global asymptotic stability of the interior equilibrium and the disease-free equilibrium of the system,respectively. According to Theorem 4.1,we can see that if the prey population is always abundant enough, the interior equilibrium of system (1.2) is globally asymptotically stable. By Theorem 4.2, we see that if the susceptible predator population S2< r2/β, that means, the susceptible predator population is not too large, then the disease-free equilibrium of system(1.2) is globally asymptotically stable. In this case, the infected predator will become extinct.

        亚洲男人的天堂av一区| 欧美一片二片午夜福利在线快 | 美女扒开屁股让男人桶| 婷婷亚洲久悠悠色悠在线播放 | 国产午夜福利精品一区二区三区 | 日本少妇被黑人xxxxx| 亚洲伊人久久大香线蕉影院| 中文字幕亚洲精品码专区| 亚洲中文字幕一区二区在线| 色窝窝无码一区二区三区| 久久久久女人精品毛片 | 国产最新一区二区三区天堂| 精品人妻一区二区三区蜜臀在线| 日本亚洲系列中文字幕| а√中文在线资源库| 久久精品国产av麻豆五月丁| 娜娜麻豆国产电影| 精品人妻无码一区二区色欲产成人| 91热国内精品永久免费观看| 亚洲一区二区三区码精品色| 精品无人区无码乱码毛片国产| 欧美猛男军警gay自慰| 精品亚洲少妇一区二区三区| 亚洲长腿丝袜中文字幕| 亚洲欧美国产精品久久| 久久精品日韩av无码| 一区二区高清视频在线观看| 国产精品日韩经典中文字幕| 国产精品熟女视频一区二区| 国产精品网站夜色| 蜜桃视频一区视频二区| 国产精品国产三级国产专播 | 欧美熟妇性xxx交潮喷| 456亚洲人成影视在线观看| 亚洲国产精品色一区二区| 色偷偷久久久精品亚洲| 国产成人啪精品视频免费软件| 美女视频很黄很a免费国产| 国产三级不卡视频在线观看| 久热国产vs视频在线观看| 国产美女在线精品亚洲二区|