2016年4期
刊物介紹
本刊主要出版來自成年神經(jīng)元的轉(zhuǎn)化與再生；損傷神經(jīng)元的軸突和樹突的修復(fù)與再生；神經(jīng)再生過程中神經(jīng)元、微環(huán)境、新生血管的變化；神經(jīng)損傷后的干預(yù)、修復(fù)、保護與再生的組織形態(tài)學(xué)變化乃至細胞生物學(xué)或細胞分子生物學(xué)的改變；神經(jīng)再生研究中的新理論、新技術(shù)、新方法。NRR雜志希望成為國際神經(jīng)再生領(lǐng)域讀者、作者和專業(yè)學(xué)者最喜歡、最認可的優(yōu)秀的專業(yè)性學(xué)術(shù)期刊。 br 期刊官方網(wǎng)站： (英文)； （中文） br 期刊投稿網(wǎng)站：
中國神經(jīng)再生研究(英文版)
- A novel technique using hydrophilic polymers to promote axonal fusion
- Examining the properties and therapeutic potential of glial restricted precursors in spinal cord injury
- The choroid plexus-cerebrospinal fluid interface in Alzheimer’s disease: more than just a barrier
- Physical interactions between activated microglia and injured axons: do all contacts lead to phagocytosis?
- Repositioning again of zonisamide for nerve regeneration
- Local translation of cell adhesion molecules in axons
- Counteraction of Nogo-A and axonal growth inhibitors by green tea polyphenols and other natural products
- Analysis of fluoxetine-induced plasticity mechanisms as a strategy for understanding plasticity related neural disorders
- Role of pleiotrophin-protein tyrosine phosphatase receptor type Z signaling in myelination
- Toll-like receptor-4 antagonism mediates benefits during neuroinflammation
- Immunomodulatory approaches to CNS injury: extracellular matrix and exosomes from extracellular matrix conditioned macrophages
- Laser therapy on points of acupuncture on nerve repair
- Prevention and protection against cerebral ischemic injury using acupuncture
- Exploring a need for improved preclinical models of post-stroke depression
- Multi-watt near-infrared light therapy as a neuroregenerative treatment for traumatic brain injury
- Neuroprotective effect of antioxidant compounds
- Autologous bone marrow mononuclear cells as neuroprotective treatment of amyotrophic lateral sclerosis
- Paracrine factors for neurodegenerative disorders: special emphasis on Parkinson’s disease
- Extrinsic and intrinsic mechanisms of axon regeneration: the need for spinal cord injury treatment strategies to address both
- Anatomical changes in the somatosensory system after large sensory loss predict strategies to promote functional recovery after spinal cord injury
- Cell proliferation and apoptosis in optic nerve and brain integration centers of adult trout Oncorhynchus mykiss after optic nerve injury
- Nerve growth factor promotes in vitro proliferation of neural stem cells from tree shrews
- Denervated hippocampus provides a favorable microenvironment for neuronal differentiation of endogenous neural stem cells
- Electroacupuncture at Baihui (DU20) acupoint upregulates mRNA expression of NeuroD molecules in the brains of newborn rats suffering in utero fetal distress
- Moxibustion upregulates hippocampal progranulin expression
- Non-viral liposome-mediated transfer of brain-derived neurotrophic factor across the blood-brain barrier
- Neuroprotective effect of Cerebralcare Granule after cerebral ischemia/reperfusion injury
- A mouse model of weight-drop closed head injury: emphasis on cognitive and neurological deficiency
- Anti-inflammatory properties of lipoxin A4 protect against diabetes mellitus complicated by focal cerebral ischemia/reperfusion injury
- Ischemic postconditioning protects against ischemic brain injury by up-regulation of acid-sensing ion channel 2a
- Correction: Enhanced motor cortex excitability after spinal cord injury
- A cascade model of information processing and encoding for retinal prosthesis
- Human umbilical cord blood-derived stem cells and brain-derived neurotrophic factor protect injured optic nerve: viscoelasticity characterization
- Senegenin inhibits neuronal apoptosis after spinal cord contusion injury
- Combined use of Y-tube conduits with human umbilical cord stem cells for repairing nerve bifurcation defects
- Supplementary motor area deactivation impacts the recovery of hand function from severe peripheral nerve injury
- Electrical stimulation of dog pudendal nerve regulates the excitatory pudendal-to-bladder reflex
- Complement components of nerve regeneration conditioned fluid influence the microenvironment of nerve regeneration
- Gait deterioration due to neural degeneration of the corticoreticular pathway: a case report